Morbus Osgood Schlatter is a juvenile osteochondria that is caratkerized by the traction of the apophytal tendon. The disease affects children and adolescents. It affects the male more often than the female sex. The prevalence is highest among sports active children. The sports where the risk of origin is increased are football, volleyball and basketball, and all sports that mean movements with frequent activation of the m. quadriceps. There are several hypotheses in relation to the reason for the occurrence, but the most accepted theory is that Morbus Osgood Schlatter affects children who have seen rapid bone growth in length, and are engaged in some of these sports. In addition to a anamnesis and physical examination, there are a number of diagnostic procedures that are used to dissester Osgood Schlatter. The gold standard is ultrasonic testing thanks to reliable results and a fine display of soft structures and easy application. The modalists of physical therapy used in the rehabilitation of Osgood Schlatter are few. This is due to sensitive areas of growth zones where the use of electroprocedures and magnetotherapy is contraindicated. In the acute stage, with regard to active inflammation, cryotherapy is applied (heat contraindicated in inflammation!). The gold standard is RICE therapy. The reduction of physical activity is recommended for all patients. The return to training and sporting life is indiviualan. The greatest consequence of therapy is abbreviation and hypotrophy and finally atrophy of inactive musculature. Therefore, despite the reduced dose of training, it is crucial to carry out stretching exercises of musculature. When the length of the muscle returns, it begins with strengthening exercises, mainly the m. quadriceps with an emphasis on the head of m. vastus medialis, but also the rest of the musculature of the lower extremity. The disease prognosis is good. The treatment is generally always conservative. In less than 2% of cases, patients undergo surgery. The disease is gradually formed, causing microtrauma on the apofizi, but after the final escalation of symptoms and stripration of the tendon the condition is further deteriorated, pain and inflammation remains. It is thought that the end of the symptoms of the disease occurs by ending the growth.